APP / NP / PA Curriculum
Level 4Hospital and Consult Cardiology

Bradycardia and Heart Block

Recognize dangerous conduction disease — the AV blocks, reversible causes, and when pacing is needed.

Advanced~24 min

Learning Objectives

  • 1.Identify the types of bradycardia and AV block.
  • 2.Recognize the high-grade blocks that are dangerous.
  • 3.Find reversible causes and know acute management/escalation.

Overview

Bradycardia ranges from benign to life-threatening. The job is recognizing high-grade conduction disease, looking for reversible causes, and escalating the symptomatic or dangerous patient.

Types

  • Sinus bradycardia — often benign (athletes, sleep, meds).
  • First-degree AV block — prolonged PR; usually benign.
  • Second-degree Mobitz I (Wenckebach) — progressive PR lengthening then a dropped beat; often benign.
  • Second-degree Mobitz II — dropped beats without PR lengthening; dangerous (can progress to complete block).
  • Third-degree (complete) heart block — AV dissociation; dangerous.

Dangerous vs benign

  • Dangerous: Mobitz II, complete heart block, and any symptomatic bradycardia (syncope, hypotension, ischemia, HF).
  • Benign: asymptomatic sinus brady, first-degree block, Mobitz I in the right context.

Reversible causes to find

  • Medications (beta-blockers, non-DHP CCBs, digoxin), ischemia/MI, hyperkalemia, hypothyroidism, increased vagal tone, hypothermia.

Acute management & escalation

  • Symptomatic/unstable bradycardia → ACLS bradycardia algorithm (atropine; transcutaneous/transvenous pacing) — physician-led.
  • Treat reversible causes (hold offending drugs, correct potassium).
  • High-grade block (Mobitz II, complete) often needs a pacemaker — cardiology/EP.

Escalate

Mobitz II, complete heart block, or symptomatic bradycardia is a physician-now situation — escalate and prepare for pacing per ACLS.

Common beginner mistakes

  • Confusing benign Mobitz I with dangerous Mobitz II.
  • Missing hyperkalemia or medication effect as a cause.
  • Not escalating symptomatic bradycardia promptly.

Mini cases

Patient with syncope; monitor shows dropped QRS complexes without preceding PR prolongation, then a run of complete dissociation.

Significance?

Show answer

Mobitz II progressing toward complete heart block — dangerous conduction disease. Escalate immediately; this often requires pacing and pacemaker evaluation.

Bradycardia at 38 with a potassium of 7.0 and peaked T waves.

Cause and action?

Show answer

Hyperkalemia-induced bradycardia — a reversible, emergent cause. Treat the hyperkalemia urgently and escalate; correcting potassium may resolve the bradycardia.

Knowledge Check Quiz

Disclaimer: This content is for educational purposes only. It is not medical advice, does not replace clinical judgment, and is not a substitute for institutional protocols or certified medical interpreters. No patient health information (PHI) should be entered into this application.